Juvenile dermatomyositis:
Occurs in childhood, as it has the same symptoms as in the shape of the electric with some difference:
- Raynaud phenomenon: rare in children.
- Muscle injury: more severe than the shape of the electric. Warning in children is better than adults.
Skin manifestations:
Topical skin manifestations are:
- Acute bluocystis, scaly papillae and pests. Appear anywhere in the skin especially on the face with swollen eyelids.
Other skin manifestations mimic lupus erythema spread and may lead to the atrophy and expansion of capillaries and subcutaneous calcification or hardening of the skin.
Skin lesions in chronic conditions are:
- Cutaneous scaly papules with enlarged capillaries and bluish atrophy similar to flat lichen, especially on the back, hands, knees and extensor surfaces.
- In parties these lesions may exhibit pigmentation and calcification.
- The mucous membranes of the mouth exhibit subcutaneous lesions.
- The scalp injury leads to non-reversible atrophic atrophy.
Muscle manifestations:
Often the muscles of the proximal limbs become swollen, painful and acidic, as the inflammatory changes end with deflationary malformations.
Systemic manifestations:
- The severity of these symptoms depends on the extent of muscle injury and include those: dyspepsia dysphagia, eye disorders, myocardial infarction, hepatic hyperplasia and lymphatic colitis.
- In-kind changes: Nectar-like profiles of cotton and bleeding appear in the ophthalmic eye examination.
Laboratory assets:
- Hypoproteinemia hyperpigmentation of leukocytes and increase the speed of stunting "ESR".
Serological tests of spastic negative in contrast to lupus erythematosus that show positive tests.
- albumin proteinuria and hematuria.
- Negative LE test.
- Tissue enzymes such as keratin, kyene, and phosphocyanase are high.
Differential diagnosis:
Systemic lupus erythematosus, scleroderma and myasthenia gravis.
Diagnosis methods:
- Clinical picture: injury to skin, muscles and other organs.
Muscle biopsy: Usually diagnosed and showing degenerative muscle changes with fragmentation, degeneration and atrophy with lymphocytes and lymphocytes.
Pathological Anatomy:
- The connective tissue thick and thick and increase in thickness.
- Collagen shows: the homogeneity of cystic fibrosis.
- Destruction of elastic fibers.
- Dwarf construction.
Treatment methods:
Treatment of dermatitis and muscle disorders is usually unsatisfactory because of the high rate of mortality the available treatments are corticosteroids and immunosuppressants.
Occurs in childhood, as it has the same symptoms as in the shape of the electric with some difference:
- Raynaud phenomenon: rare in children.
- Muscle injury: more severe than the shape of the electric. Warning in children is better than adults.
Skin manifestations:
Topical skin manifestations are:
- Acute bluocystis, scaly papillae and pests. Appear anywhere in the skin especially on the face with swollen eyelids.
Other skin manifestations mimic lupus erythema spread and may lead to the atrophy and expansion of capillaries and subcutaneous calcification or hardening of the skin.
Skin lesions in chronic conditions are:
- Cutaneous scaly papules with enlarged capillaries and bluish atrophy similar to flat lichen, especially on the back, hands, knees and extensor surfaces.
- In parties these lesions may exhibit pigmentation and calcification.
- The mucous membranes of the mouth exhibit subcutaneous lesions.
- The scalp injury leads to non-reversible atrophic atrophy.
Muscle manifestations:
Often the muscles of the proximal limbs become swollen, painful and acidic, as the inflammatory changes end with deflationary malformations.
Systemic manifestations:
- The severity of these symptoms depends on the extent of muscle injury and include those: dyspepsia dysphagia, eye disorders, myocardial infarction, hepatic hyperplasia and lymphatic colitis.
- In-kind changes: Nectar-like profiles of cotton and bleeding appear in the ophthalmic eye examination.
Laboratory assets:
- Hypoproteinemia hyperpigmentation of leukocytes and increase the speed of stunting "ESR".
Serological tests of spastic negative in contrast to lupus erythematosus that show positive tests.
- albumin proteinuria and hematuria.
- Negative LE test.
- Tissue enzymes such as keratin, kyene, and phosphocyanase are high.
Differential diagnosis:
Systemic lupus erythematosus, scleroderma and myasthenia gravis.
Diagnosis methods:
- Clinical picture: injury to skin, muscles and other organs.
Muscle biopsy: Usually diagnosed and showing degenerative muscle changes with fragmentation, degeneration and atrophy with lymphocytes and lymphocytes.
Pathological Anatomy:
- The connective tissue thick and thick and increase in thickness.
- Collagen shows: the homogeneity of cystic fibrosis.
- Destruction of elastic fibers.
- Dwarf construction.
Treatment methods:
Treatment of dermatitis and muscle disorders is usually unsatisfactory because of the high rate of mortality the available treatments are corticosteroids and immunosuppressants.
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skin diseases